Correlation between mycotoxins and OAT fungal metabolites

[Suzanne Ellison]

What conclusions might you draw in a case like the following?

Initial OAT with high arabinose, tartaric and furancaarbonylglycine, and oxalate elevated also but negative mycotoxins (completely absent).
Patient did not tolerate glutathione, phosphatidylcholine, or even NAC initially. Treated for yeast, had a Herx reaction for the first two days. AFter 2 weeks of that, put on silymarin, and complete detox supplementation, a product from nutritionalspecialties with at least some NAC…this was tolerated.

Sauna almost daily for months. Assume no dietary exposures due to a very clean diet.

Repeat the mycotoxin and OAT: all the fungal markers cleared except slightly elevated arabinose and oxalate.
And 4 of the mycotoxins are very positive now.

We will be checking the patients spaces for mold…and add binders.

So, do you see this? Is the mold likely in the microbiome or sinuses? Or do you think that mycotoxins, separate from a current infestation of the mold itself, will store up in fat and other tissues, being released when the detoxifying ability increases?

Can the improvement in the fungal markers, in spite of the positive mycotoxins, mean that the mold is less likely to be infesting the patient?

Topic

[Kurt Woeller]

Suzanne,
Very interesting case!

The normalization of the Furancarbonylglycine means most likely the Aspergillus mold is not in the digestive system anymore. It’s always possible its in the sinuses, but your would expect some sinus/respiratory problems, e.g. congestion, cough.

The mycotoxins are lipophilic, so it is possible for them to be stored in the fat and the release through lipolysis. Sauna is known to induce fat toxin release and then excretion through the urine. Glutathione and NAC will mobilize lipophilic mycotoxins too.

Dr. Woeller

[Author]

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